The Hormone of Chads - [ULTIMATE Guide to DHT Optimization]
What if everything you thought you knew about male hormones was a lie?
What if doctors have been prescribing the wrong hormones for male HRT for decades?
What if most of the benefits we associate with testosterone are actually from its metabolite Dihydrotestosterone (DHT)?
DHT, the hormone of chads, is produced when a free testosterone molecule interacts with the 5AR (5-alpha reductase) enzyme.
Now suppose this hormone determined:
The prominence of your masculine physical features?
The extent of your masculine behavioral traits?
Your “eggplant” size and development?
Your level of physical attractiveness to women?
Your level of confidence and personal conviction?
Your physical strength and speed capabilities?
Your ability to remain calm under stress?
The ability to communicate directly and assertively?
The upper limit of your spatial reasoning skills?
Would it not make sense to pay attention to it? Talk about it? Optimize it?
Of course, it would. So, why aren’t we?
Let’s find out:
Deep Soy’s Attack on Masculinity
It’s no conspiracy that DHT is rapidly declining each decade, greatly exceeding the well-documented decline in testosterone levels.
Besides the fact that you can walk outside and notice that the young men of today look nothing like the young men of your grandfather’s generation, you can see a massive difference in the documented average DHT levels in studies of the past, compared with today’s reference range.
Are we supposed to ignore the fact that AVERAGE man in 1982 had nearly THREE TIMES HIGHER DHT levels than today? That any average man would be flagged as HIGH today?
Are we supposed to accept the modern reference range that tells us a man & a woman can have the same DHT levels (12-19ng/dl) and both are NORMAL?
The hormone that makes a man a man. The hormone without which you would have been assigned female at birth. We’re really supposed to accept that men and women should have the same levels?
A Medical Miracle Memory-Holed
DHT isn’t a new discovery by any means. It has existed in mammals for millions of years, discovered and synthesized in 1935, and introduced as the prescription drug Neodrol in 1953. Neodrol was FDA-approved and prescribed as Androgen Replacement Therapy for hypogonadal men, but it was silently taken off the market sometime in the 60s and not much else is known about it. The last time it was mentioned was in the American Medical Association publication “New and Nonofficial Drugs” in 1964.
You may be wondering why it was pulled off the market, and there are likely a few factors involved:
DHT cannot function as standalone hormone replacement therapy. Testosterone is still necessary due to its unique modulatory effects of various systems within the body and its conversion to estradiol and other minor androgens. DHT solo = terrible side effects.
Pharmaceutical companies were looking to create more profitable, patented Testosterone and DHT derivatives. Around this time Metandienone (Dbol), Fluoxymesterone (Halotestin), Drostanolone (Masteron), Methenolone (Primobolin), Nandrolone (Deca), and Methyltestosterone were introduced and marketed aggressively. Even though they could not fully replace the functions of testosterone and DHT and had tons of side effects, the medical industry went all in on synthetics. These synthetic androgens would later become cherished by bodybuilders and abandoned by the medical industry.
Fun fact: JFK was taking the synthetic androgens Fluoxymesterone & Methyltestosterone as part of his hormone replacement therapy & addison’s disease management protocol.
Deep Soy Strikes
While DHT remained forgotten in the US, it was scarcely prescribed in other parts of the world, including the UK and France. Things would soon take an ugly turn for DHT in the 80s when poorly designed studies began identifying DHT as the root cause of benign prostatic hyperplasia (BPH) and prostate cancer.
Merck quickly jumped to developing 5AR inhibitors to reduce testosterone’s conversion to DHT and Finasteride was approved for BPH in 1992. While inhibiting 5AR does indeed resolve some cases of BPH, we would later learn that it is nowhere near the most effective pathway to treat BPH. Regardless of effectiveness, Merck marketed finasteride aggressively, which required doctors to be re-educated and medical school curriculums to be revised. This was just the beginning of the smear campaign that would take place. Other poorly designed studies began to take place that identified DHT as the root cause of hair loss (it’s not) and Merck was now sitting on a multibillion dollar gold-mine of a patent. In 1997, Finasteride was approved for male-pattern-baldness and the indoctrination of medical students and brainwashing of doctors intensified.
The paradigm shifted from “DHT is an essential hormone for male health, development, and quality of life” to “DHT is a useless hormone that is only required for puberty and it must be eliminated to reduce BPH, prostate, cancer, and hair loss.”
This paradigm shift has brought us to this point in time, where DHT levels are mysteriously declining and men are being deprived of the knowledge and resources they need to claim their birthright of achieving peak masculine excellence. It’s time to change that.
Physical Benefits of DHT
The following information has been synthesized from numerous studies on DHT as well as the reports of 35 underground biohackers who underwent 6+ months of experiments with exogenous DHT esters at multiple supraphysiological dosages and combinations with testosterone, hCG, DHEA, pregnenolone, and estradiol.
DHT binds to androgen receptors with approximately 4-5 times higher affinity than testosterone. Milligram for milligram, it is roughly as potent as the infamous Trenbolone.
Potently increases muscle mass
Increases muscle density/hardness
Increases fast-twitch fiber activation (more strength, speed)
Increases physical energy & stamina
Promotes an increase in 🍆 length and girth, including in adults
Reduces gynecomastia (bitch tits) [Source]
Reduces water retention and blood pressure through mineralocorticoid receptor antagonism (more dry and shredded look) Sources: [1], [2]
Potently burns fat through cortisol suppression and metabolic rate increase Sources: [1], [2], [3]
Potently preserves muscle mass in caloric deficits
Decreases vocal pitch (deepens voice); Men with lower voices, on average, earn more and are perceived as more attractive and trustworthy. Sources: [1], [2], [3].
Increases facial hair growth and density
Increases the production of pheromones, which increase a man’s perceived attractiveness, trustworthiness, and dominance.
Reduces risk of heart disease
Example of a high testosterone vs high testosterone and DHT physique. Yes, I realize that this is a picture of Connor Murphy that was taken on the same day, but it stands as a good example of how DHT increases muscle definition, density/hardness, and produces a “dry” and lean appearance, through its unique activity at AR/MR and its modulatory effect on cortisol.
Mental Benefits of DHT
Drastic increase in stress tolerance; calming & anxiolytic without sedation.
Increase in pain tolerance
Dramatic increase in self-confidence and conviction
Significant improvement in cognitive performance, particularly spatial reasoning, logical ability, and memory
Anti-depressive/mood improvement
Increase in energy, motivation, and drive
Libido enhancement
It’s important to note that just like testosterone converts to DHT, DHT converts to & influences the production of other hormones known as neurosteroids AKA “brain steroids” These neurosteroids are where the majority of the mental benefits of DHT come from.
3a-Androstanediol: weak androgen agonist, weakERβ agonist, and potent GABA_A Positive Allosteric Modulator (think endogenous “Xanax” without the intoxication)
Potently reduces anxiety, reduces anxious behavior, and improves cognitive performance under stress.
Reduces perception of pain
Increases libido
Reduces convulsive activity and glutamate signaling
Enhances focus, motivation, and reward signaling (dopaminergic)
Enhances memory
Neuroprotective
Modulates locomotor activity
Androsterone: mild androgen agonist, weak ERβ agonist, GABA_A Positive Allosteric Modulator (think endogenous “Xanax” without the intoxication), and pheromone
Moderately reduces anxiety, reduces anxious behavior, and improves cognitive performance under stress.
Reduces perception of pain
Reduces convulsive activity and glutamate signaling
Enhances focus, motivation, and reward signaling dopaminergic)
Enhances memory
Neuroprotective
Modulates locomotor activity
3β-Androstanediol: weak androgen agonist, potent ERβ agonist
Libido regulation
Anti-depressant properties
Decreases perception of pain
Neuroprotective
Modulates locomotor activity
Epi-Androsterone: Opioid receptor modulator, FXR agonist, PPARγ agonist, and EP agonist.
Anticonvulsant properties
Opioid-like effects: analgesia, euphoria, hyperactivity, etc
Enhances memory
Neuroprotective
Modulates locomotor activity
It is no coincidence that with the decline of these neurosteroids, we’ve seen male depression and anxiety skyrocket over the decades. We’ve gone from men fearlessly facing death on the frontlines of war to getting PTSD from “cyberbullying”. From cold approaching as the norm to being too afraid to interact with females outside the DMs. We went from long-brutal days in the coal mines without a single complaint, to taking “mental health days” from cushy desk jobs because they’re “triggering anxiety”.
While the modern low DHT man makes a great tax servant for the ruling class, he is unable to be the superhuman his family and future generations need him to be. Enough is enough. It’s time to start talking about DHT Maxxing.